PDA_PROX
Technical documentation
Posterior / inferoseptal infarction (proximal PDA)
Occlusion of the posterior descending artery in the posterior interventricular groove. Inferoseptal and posterior infarction. In right dominance it arises from RCA; in left dominance from distal LCx. Frank elevation in V7-V9 and inferior leads, with anterior mirror image in V1-V3.
Also known as: Posterior / inferoseptal infarction (proximal PDA) · descendente posterior · PDA · arteria interventricular posterior · IVP · infarto posterior · infarto inferoseptal · infarto infero-septal · SCACEST posterior · posterior descending artery · posterior STEMI · PDA STEMI
What parameters does the model use?
The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.
What parameters does the model use?
| Parameter | Model value |
|---|---|
| Involved artery | RCA |
| ST elevation in | V7, V8, V9, III, aVF, II |
| Reciprocal depression in | V1, V2, V3 |
| AHA 17-segment model | 3, 4, 9, 10, 15 |
| Occlusion severity | Total |
Which leads should be evaluated?
The catalog marks as primary diagnostic leads V7, V8, V9, III, aVF, II. ST elevation in V7, V8, V9, III, aVF, II; reciprocal ST depression in V1, V2, V3.
In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.
Frequently asked clinical questions
How is Posterior / inferoseptal infarction (proximal PDA) identified on the 12-lead ECG?
Occlusion of the posterior descending artery in the posterior interventricular groove. Inferoseptal and posterior infarction. In right dominance it arises from RCA; in left dominance from distal LCx. Frank elevation in V7-V9 and inferior leads, with anterior mirror image in V1-V3. The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.
Which leads are critical for diagnostic confirmation?
The primary diagnostic leads are V7, V8, V9, III, aVF, II. ST elevation in V7, V8, V9, III, aVF, II; reciprocal ST depression in V1, V2, V3. In the simulator, these channels highlight the correlation with anatomical vectors.
What is the underlying electrophysiological or anatomical mechanism?
It represents transmural ischemia secondary to occlusion in the territory of the RCA.
How can this condition be explored in the 3D simulator?
MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.
How do you open this configuration?
The simulator encodes its full state in the query string, providing a stable and reproducible URL:
https://mioatlas.com/simulador/en/?syn=SCACEST&ter=PDA_PROX&add=1
Related entities
Other entities in the same category within the catalog:
Sources and bibliographic references
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Wagner GS, Macfarlane P, Wellens H, et al. (2009). "AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram: Part VI: Acute ischemia/infarction". Circulation.
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Cerqueira MD, Weissman NJ, Dilsizian V, et al. (2002). "Standardized myocardial segmentation and nomenclature for tomographic imaging of the heart". Circulation.
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Thygesen K, Alpert JS, Jaffe AS, et al. (2019). "Fourth Universal Definition of Myocardial Infarction (2018)". Eur Heart J.