PEA
Technical documentation
Pulseless Electrical Activity
Presence of organized electrical activity on ECG (sinus, bradycardia, bundle branch block, or other rhythms) in a patient with no palpable arterial pulse and no effective cardiac output. Caused by reversible profound mechanical or metabolic collapse (hypovolemia, hypoxia, tension pneumothorax, tamponade, pulmonary embolism, acidosis).
Also known as: Pulseless Electrical Activity · actividad electrica sin pulso · actividad eléctrica sin pulso · AESP · disociacion electromecanica · pulseless electrical activity · PEA · DEM
What parameters does the model use?
The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.
What parameters does the model use?
| Parameter | Model value |
|---|---|
| Mechanism | Mixto |
| Modeled rate | 20–60 bpm (default 38) |
| Regularity | Regular |
| P wave | Absent |
| QRS duration | 165 ms |
| QRS morphology | Wide, ventricular origin |
| AV conduction | No conduction to ventricles |
| Severity | Life-threatening |
Which leads should be evaluated?
The catalog marks as primary diagnostic leads II. Organized electrical complexes on ECG without detectable pulse or mechanical cardiac output.
In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.
Frequently asked clinical questions
How is Pulseless Electrical Activity identified on the 12-lead ECG?
Presence of organized electrical activity on ECG (sinus, bradycardia, bundle branch block, or other rhythms) in a patient with no palpable arterial pulse and no effective cardiac output. Caused by reversible profound mechanical or metabolic collapse (hypovolemia, hypoxia, tension pneumothorax, tamponade, pulmonary embolism, acidosis). The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.
Which leads are critical for diagnostic confirmation?
The primary diagnostic leads are II. Organized electrical complexes on ECG without detectable pulse or mechanical cardiac output. In the simulator, these channels highlight the correlation with anatomical vectors.
What is the underlying electrophysiological or anatomical mechanism?
It originates from altered conduction in the cardiac conduction system, mediated by mixto.
How can this condition be explored in the 3D simulator?
MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.
How do you open this configuration?
The simulator encodes its full state in the query string, providing a stable and reproducible URL:
https://mioatlas.com/simulador/en/?mode=conduction&arr=PEA
Related entities
Other entities in the same category within the catalog:
Sources and bibliographic references
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Anderson RH, Yanni J, Boyett MR, Chandler NJ, Dobrzynski H (2009). "The anatomy of the cardiac conduction system". Clin Anat.
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Kligfield P, Gettes LS, Bailey JJ, et al. (2007). "Recommendations for the standardization and interpretation of the electrocardiogram: Part I: The electrocardiogram and its technology". Circulation.
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Frank E (1956). "An accurate, clinically practical system for spatial vectorcardiography". Circulation.