Left Anterior Descending (LAD) LAD_WRAPAROUND Technical documentation

Apical-inferior infarction (recurrent / wraparound LAD)

Type III LAD anatomy (long / wrapping): wraps around the apex and supplies the lower third of the posterior groove. Causes simultaneous anterior (V3-V5) and inferior (II, III, aVF with II > III) ST elevation, with characteristic reciprocal depression in aVL.

Generated from the computational engine catalog · Educational reference without specialist cardiology review.

Also known as: Apical-inferior infarction (recurrent / wraparound LAD) · DA recurrente · LAD wraparound · infarto apico-inferior · infarto apicoinferior · LAD tipo III · wraparound LAD · apical inferior STEMI

What parameters does the model use?

The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.

What parameters does the model use?

Parameter Model value
Involved artery LAD
ST elevation in V3, V4, V5, II, III, aVF
Reciprocal depression in aVL
AHA 17-segment model 13, 14, 15, 17
Occlusion severity Total

Which leads should be evaluated?

The catalog marks as primary diagnostic leads V3, V4, V5, II, III, aVF. ST elevation in V3, V4, V5, II, III, aVF; reciprocal ST depression in aVL.

In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.

Frequently asked clinical questions

How is Apical-inferior infarction (recurrent / wraparound LAD) identified on the 12-lead ECG?

Type III LAD anatomy (long / wrapping): wraps around the apex and supplies the lower third of the posterior groove. Causes simultaneous anterior (V3-V5) and inferior (II, III, aVF with II > III) ST elevation, with characteristic reciprocal depression in aVL. The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.

Which leads are critical for diagnostic confirmation?

The primary diagnostic leads are V3, V4, V5, II, III, aVF. ST elevation in V3, V4, V5, II, III, aVF; reciprocal ST depression in aVL. In the simulator, these channels highlight the correlation with anatomical vectors.

What is the underlying electrophysiological or anatomical mechanism?

It represents transmural ischemia secondary to occlusion in the territory of the LAD.

How can this condition be explored in the 3D simulator?

MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.

How do you open this configuration?

The simulator encodes its full state in the query string, providing a stable and reproducible URL:

https://mioatlas.com/simulador/en/?syn=SCACEST&ter=LAD_WRAPAROUND

Related entities

Other entities in the same category within the catalog:

Sources and bibliographic references

  1. Wagner GS, Macfarlane P, Wellens H, et al. (2009). "AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram: Part VI: Acute ischemia/infarction". Circulation.
  2. Cerqueira MD, Weissman NJ, Dilsizian V, et al. (2002). "Standardized myocardial segmentation and nomenclature for tomographic imaging of the heart". Circulation.
  3. Thygesen K, Alpert JS, Jaffe AS, et al. (2019). "Fourth Universal Definition of Myocardial Infarction (2018)". Eur Heart J.