LCX_PROX
Technical documentation
Lateral and posterolateral infarction (proximal LCx)
Proximal circumflex occlusion before OM1. In right dominance it produces posterolateral infarction (I, aVL, V5-V6, V7-V9) with anterior mirror changes in V1-V3. In left dominance it additionally compromises the complete inferior wall (II, III, aVF with II >= III).
Also known as: Lateral and posterolateral infarction (proximal LCx) · circunfleja proximal · CX proximal · LCx proximal · infarto lateral · SCACEST posterolateral · lateral STEMI · proximal circumflex occlusion · proximal LCx STEMI
What parameters does the model use?
The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.
What parameters does the model use?
| Parameter | Model value |
|---|---|
| Involved artery | LCX |
| ST elevation in | I, aVL, V5, V6, V7, V8, V9 |
| Reciprocal depression in | V1, V2, V3 |
| AHA 17-segment model | 5, 6, 11, 12, 16 |
| Occlusion severity | Total |
Which leads should be evaluated?
The catalog marks as primary diagnostic leads I, aVL, V5, V6, V7, V8, V9. ST elevation in I, aVL, V5, V6, V7, V8, V9; reciprocal ST depression in V1, V2, V3.
In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.
Frequently asked clinical questions
How is Lateral and posterolateral infarction (proximal LCx) identified on the 12-lead ECG?
Proximal circumflex occlusion before OM1. In right dominance it produces posterolateral infarction (I, aVL, V5-V6, V7-V9) with anterior mirror changes in V1-V3. In left dominance it additionally compromises the complete inferior wall (II, III, aVF with II >= III). The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.
Which leads are critical for diagnostic confirmation?
The primary diagnostic leads are I, aVL, V5, V6, V7, V8, V9. ST elevation in I, aVL, V5, V6, V7, V8, V9; reciprocal ST depression in V1, V2, V3. In the simulator, these channels highlight the correlation with anatomical vectors.
What is the underlying electrophysiological or anatomical mechanism?
It represents transmural ischemia secondary to occlusion in the territory of the LCX.
How can this condition be explored in the 3D simulator?
MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.
How do you open this configuration?
The simulator encodes its full state in the query string, providing a stable and reproducible URL:
https://mioatlas.com/simulador/en/?syn=SCACEST&ter=LCX_PROX&add=1
Related entities
Other entities in the same category within the catalog:
Sources and bibliographic references
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Wagner GS, Macfarlane P, Wellens H, et al. (2009). "AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram: Part VI: Acute ischemia/infarction". Circulation.
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Cerqueira MD, Weissman NJ, Dilsizian V, et al. (2002). "Standardized myocardial segmentation and nomenclature for tomographic imaging of the heart". Circulation.
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Thygesen K, Alpert JS, Jaffe AS, et al. (2019). "Fourth Universal Definition of Myocardial Infarction (2018)". Eur Heart J.