SINUS_ARRHYTHMIA
Technical documentation
Respiratory Sinus Arrhythmia
Physiological heart rate variation linked to the respiratory cycle: accelerates during inspiration (vagal withdrawal) and decelerates during expiration (increased vagal tone). Completely benign, common in young and healthy individuals. P wave morphology and PR interval remain identical in all cycles.
Also known as: Respiratory Sinus Arrhythmia · arritmia sinusal · arritmia sinusal fasica · arritmia sinusal respiratoria · respiratory sinus arrhythmia · sinus arrhythmia
What parameters does the model use?
The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.
What parameters does the model use?
| Parameter | Model value |
|---|---|
| Mechanism | Automatismo |
| Impulse origin | Sinoatrial node (SA node) |
| Modeled rate | 55–95 bpm (default 72) |
| Regularity | Regularly irregular |
| P wave | Sinus (upright in I, II, aVF) |
| PR interval | 120–200 ms (default 160) |
| QRS duration | 90 ms |
| QRS morphology | Normal (narrow) |
| AV conduction | 1:1 AV conduction |
| Severity | Benign |
Which leads should be evaluated?
The catalog marks as primary diagnostic leads II. Respiratory variation in RR intervals on lead II rhythm strip.
In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.
Frequently asked clinical questions
How is Respiratory Sinus Arrhythmia identified on the 12-lead ECG?
Physiological heart rate variation linked to the respiratory cycle: accelerates during inspiration (vagal withdrawal) and decelerates during expiration (increased vagal tone). Completely benign, common in young and healthy individuals. P wave morphology and PR interval remain identical in all cycles. The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.
Which leads are critical for diagnostic confirmation?
The primary diagnostic leads are II. Respiratory variation in RR intervals on lead II rhythm strip. In the simulator, these channels highlight the correlation with anatomical vectors.
What is the underlying electrophysiological or anatomical mechanism?
It originates from altered conduction in the SA_NODE, mediated by automatismo.
How can this condition be explored in the 3D simulator?
MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.
How do you open this configuration?
The simulator encodes its full state in the query string, providing a stable and reproducible URL:
https://mioatlas.com/simulador/en/?mode=conduction&arr=SINUS_ARRHYTHMIA
Related entities
Other entities in the same category within the catalog:
Sources and bibliographic references
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Anderson RH, Yanni J, Boyett MR, Chandler NJ, Dobrzynski H (2009). "The anatomy of the cardiac conduction system". Clin Anat.
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Kligfield P, Gettes LS, Bailey JJ, et al. (2007). "Recommendations for the standardization and interpretation of the electrocardiogram: Part I: The electrocardiogram and its technology". Circulation.
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Rautaharju PM, Surawicz B, Gettes LS, et al. (2009). "AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram: Part IV: The ST segment, T and U waves, and the QT interval". Circulation.