SINUS_TACHY
Technical documentation
Sinus Tachycardia
Sinus rhythm at a rate above 100 bpm. Typical physiological response to exercise, fever, hypovolemia, anemia, hyperthyroidism, pain, anxiety, or adrenergic stimulation. Normal P wave morphology, though at very fast rates it may merge with the preceding T wave.
Also known as: Sinus Tachycardia · taquicardia sinusal · sinus tachycardia · sinus tachy · TS
What parameters does the model use?
The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.
What parameters does the model use?
| Parameter | Model value |
|---|---|
| Mechanism | Automatismo |
| Impulse origin | Sinoatrial node (SA node) |
| Modeled rate | 101–170 bpm (default 125) |
| Regularity | Regular |
| P wave | Sinus (upright in I, II, aVF) |
| PR interval | 110–160 ms (default 135) |
| QRS duration | 88 ms |
| QRS morphology | Normal (narrow) |
| AV conduction | 1:1 AV conduction |
| Severity | Benign |
Which leads should be evaluated?
The catalog marks as primary diagnostic leads II. Rhythm strip (lead II): sinus P wave at > 100 bpm.
In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.
Frequently asked clinical questions
How is Sinus Tachycardia identified on the 12-lead ECG?
Sinus rhythm at a rate above 100 bpm. Typical physiological response to exercise, fever, hypovolemia, anemia, hyperthyroidism, pain, anxiety, or adrenergic stimulation. Normal P wave morphology, though at very fast rates it may merge with the preceding T wave. The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.
Which leads are critical for diagnostic confirmation?
The primary diagnostic leads are II. Rhythm strip (lead II): sinus P wave at > 100 bpm. In the simulator, these channels highlight the correlation with anatomical vectors.
What is the underlying electrophysiological or anatomical mechanism?
It originates from altered conduction in the SA_NODE, mediated by automatismo.
How can this condition be explored in the 3D simulator?
MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.
How do you open this configuration?
The simulator encodes its full state in the query string, providing a stable and reproducible URL:
https://mioatlas.com/simulador/en/?mode=conduction&arr=SINUS_TACHY
Related entities
Other entities in the same category within the catalog:
Sources and bibliographic references
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Anderson RH, Yanni J, Boyett MR, Chandler NJ, Dobrzynski H (2009). "The anatomy of the cardiac conduction system". Clin Anat.
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Kligfield P, Gettes LS, Bailey JJ, et al. (2007). "Recommendations for the standardization and interpretation of the electrocardiogram: Part I: The electrocardiogram and its technology". Circulation.
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Rautaharju PM, Surawicz B, Gettes LS, et al. (2009). "AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram: Part IV: The ST segment, T and U waves, and the QT interval". Circulation.