Preexcitation AVRT_ANTIDROMIC Technical documentation

Antidromic AV Reentrant Tachycardia

Less common form of AVRT in WPW (< 10%). The reentrant circuit conducts anterogradely down the accessory pathway (producing maximal preexcitation and very wide, bizarre QRS) and retrogradely up the AV node or a second accessory pathway. Can be difficult to distinguish on ECG from ventricular tachycardia.

Generated from the computational engine catalog · Educational reference without specialist cardiology review.

Also known as: Antidromic AV Reentrant Tachycardia · taquicardia antidromica · reentrada AV antidromica · TRAV antidromica · taquicardia por reentrada AV antidromica · antidromic AVRT · antidromic reciprocating tachycardia

What parameters does the model use?

The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.

What parameters does the model use?

Parameter Model value
Mechanism Reentrada
Impulse origin Accessory pathway (Bundle of Kent)
Modeled rate 150–250 bpm (default 190)
Regularity Regular
P wave Retrograde
QRS duration 160 ms
QRS morphology With delta wave (preexcitation)
AV conduction 1:1 AV conduction
Severity Life-threatening

Which leads should be evaluated?

The catalog marks as primary diagnostic leads V1, V2, V3, V4, V5, V6. Wide maximally preexcited QRS complexes across all precordial leads.

In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.

Frequently asked clinical questions

How is Antidromic AV Reentrant Tachycardia identified on the 12-lead ECG?

Less common form of AVRT in WPW (< 10%). The reentrant circuit conducts anterogradely down the accessory pathway (producing maximal preexcitation and very wide, bizarre QRS) and retrogradely up the AV node or a second accessory pathway. Can be difficult to distinguish on ECG from ventricular tachycardia. The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.

Which leads are critical for diagnostic confirmation?

The primary diagnostic leads are V1, V2, V3, V4, V5, V6. Wide maximally preexcited QRS complexes across all precordial leads. In the simulator, these channels highlight the correlation with anatomical vectors.

What is the underlying electrophysiological or anatomical mechanism?

It originates from altered conduction in the ACCESSORY_KENT, mediated by reentrada.

How can this condition be explored in the 3D simulator?

MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.

How do you open this configuration?

The simulator encodes its full state in the query string, providing a stable and reproducible URL:

https://mioatlas.com/simulador/en/?mode=conduction&arr=AVRT_ANTIDROMIC

Related entities

Other entities in the same category within the catalog:

Sources and bibliographic references

  1. Anderson RH, Yanni J, Boyett MR, Chandler NJ, Dobrzynski H (2009). "The anatomy of the cardiac conduction system". Clin Anat.
  2. Surawicz B, Childers R, Deal BJ, et al. (2009). "AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram: Part III: Intraventricular conduction disturbances". Circulation.
  3. Kligfield P, Gettes LS, Bailey JJ, et al. (2007). "Recommendations for the standardization and interpretation of the electrocardiogram: Part I: The electrocardiogram and its technology". Circulation.