AFIB_WPW
Technical documentation
Preexcited Atrial Fibrillation
Extremely dangerous emergency: chaotic atrial fibrillation impulses conduct anterogradely down an accessory pathway that lacks the decremental conduction properties of the AV node. Produces an irregularly irregular wide-QRS tachycardia at extremely rapid ventricular rates (> 200-300 bpm), with risk of degenerating into ventricular fibrillation. AV nodal blockers (adenosine, beta-blockers, verapamil, digoxin) are strictly contraindicated.
Also known as: Preexcited Atrial Fibrillation · fibrilacion auricular preexcitada · FA preexcitada · FA en WPW · pre-excited atrial fibrillation · preexcited AFIB · FBI tachycardia
What parameters does the model use?
The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.
What parameters does the model use?
| Parameter | Model value |
|---|---|
| Mechanism | Mixto |
| Modeled rate | 180–300 bpm (default 240) |
| Regularity | Irregular |
| P wave | Fibrillatory waves (f waves) |
| QRS duration | 170 ms |
| QRS morphology | With delta wave (preexcitation) |
| AV conduction | Variable |
| Severity | Life-threatening |
Which leads should be evaluated?
The catalog marks as primary diagnostic leads V1, V2, V3, V4, V5, V6, II. Irregularly irregular wide-complex tachycardia with beat-to-beat variation in QRS width.
In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.
Frequently asked clinical questions
How is Preexcited Atrial Fibrillation identified on the 12-lead ECG?
Extremely dangerous emergency: chaotic atrial fibrillation impulses conduct anterogradely down an accessory pathway that lacks the decremental conduction properties of the AV node. Produces an irregularly irregular wide-QRS tachycardia at extremely rapid ventricular rates (> 200-300 bpm), with risk of degenerating into ventricular fibrillation. AV nodal blockers (adenosine, beta-blockers, verapamil, digoxin) are strictly contraindicated. The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.
Which leads are critical for diagnostic confirmation?
The primary diagnostic leads are V1, V2, V3, V4, V5, V6, II. Irregularly irregular wide-complex tachycardia with beat-to-beat variation in QRS width. In the simulator, these channels highlight the correlation with anatomical vectors.
What is the underlying electrophysiological or anatomical mechanism?
It originates from altered conduction in the cardiac conduction system, mediated by mixto.
How can this condition be explored in the 3D simulator?
MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.
How do you open this configuration?
The simulator encodes its full state in the query string, providing a stable and reproducible URL:
https://mioatlas.com/simulador/en/?mode=conduction&arr=AFIB_WPW
Related entities
Other entities in the same category within the catalog:
Sources and bibliographic references
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Anderson RH, Yanni J, Boyett MR, Chandler NJ, Dobrzynski H (2009). "The anatomy of the cardiac conduction system". Clin Anat.
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Surawicz B, Childers R, Deal BJ, et al. (2009). "AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram: Part III: Intraventricular conduction disturbances". Circulation.
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Kligfield P, Gettes LS, Bailey JJ, et al. (2007). "Recommendations for the standardization and interpretation of the electrocardiogram: Part I: The electrocardiogram and its technology". Circulation.