MAT
Technical documentation
Multifocal Atrial Tachycardia
Supraventricular tachycardia characterized by ≥ 3 distinct P wave morphologies in the same lead, with irregular PP, PR, and RR intervals. Typical in elderly patients with acute pulmonary disease exacerbation (COPD), hypoxia, hypercapnia, or theophylline toxicity.
Also known as: Multifocal Atrial Tachycardia · taquicardia auricular multifocal · taquicardia auricular caotica · multifocal atrial tachycardia · MAT · TAM
What parameters does the model use?
The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.
What parameters does the model use?
| Parameter | Model value |
|---|---|
| Mechanism | Automatismo |
| Modeled rate | 100–180 bpm (default 130) |
| Regularity | Irregular |
| P wave | Multifocal (≥ 3 morphologies) |
| PR interval | 100–220 ms (default 150) |
| QRS duration | 90 ms |
| QRS morphology | Normal (narrow) |
| AV conduction | Variable |
| Severity | Clinically significant |
Which leads should be evaluated?
The catalog marks as primary diagnostic leads II, V1. Three or more distinct P wave morphologies with variable PP/PR intervals in II and V1.
In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.
Frequently asked clinical questions
How is Multifocal Atrial Tachycardia identified on the 12-lead ECG?
Supraventricular tachycardia characterized by ≥ 3 distinct P wave morphologies in the same lead, with irregular PP, PR, and RR intervals. Typical in elderly patients with acute pulmonary disease exacerbation (COPD), hypoxia, hypercapnia, or theophylline toxicity. The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.
Which leads are critical for diagnostic confirmation?
The primary diagnostic leads are II, V1. Three or more distinct P wave morphologies with variable PP/PR intervals in II and V1. In the simulator, these channels highlight the correlation with anatomical vectors.
What is the underlying electrophysiological or anatomical mechanism?
It originates from altered conduction in the cardiac conduction system, mediated by automatismo.
How can this condition be explored in the 3D simulator?
MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.
How do you open this configuration?
The simulator encodes its full state in the query string, providing a stable and reproducible URL:
https://mioatlas.com/simulador/en/?mode=conduction&arr=MAT
Related entities
Other entities in the same category within the catalog:
Sources and bibliographic references
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Anderson RH, Yanni J, Boyett MR, Chandler NJ, Dobrzynski H (2009). "The anatomy of the cardiac conduction system". Clin Anat.
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Kligfield P, Gettes LS, Bailey JJ, et al. (2007). "Recommendations for the standardization and interpretation of the electrocardiogram: Part I: The electrocardiogram and its technology". Circulation.
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Sovilj S, Magjarevic R, Lovell NH, Dokos S (2013). "A simplified 3D model of whole heart electrical activity and 12-lead ECG generation". Comput Math Methods Med.