VT_POLYMORPHIC
Technical documentation
Polymorphic Ventricular Tachycardia
Ventricular tachycardia with beat-to-beat variation in QRS morphology and axis. Commonly triggered by acute myocardial ischemia (coronary occlusion), severe electrolyte abnormalities, or catecholaminergic surge. Rapidly deteriorates into ventricular fibrillation and hemodynamic collapse.
Also known as: Polymorphic Ventricular Tachycardia · taquicardia ventricular polimorfa · taquicardia ventricular polimorfica · TV polimorfa · polymorphic ventricular tachycardia · polymorphic VT · PVT
What parameters does the model use?
The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.
What parameters does the model use?
| Parameter | Model value |
|---|---|
| Mechanism | Reentrada |
| Modeled rate | 150–250 bpm (default 200) |
| Regularity | Irregular |
| P wave | Dissociated from QRS |
| QRS duration | 170 ms |
| QRS morphology | Polymorphic |
| AV conduction | Atrioventricular dissociation |
| Severity | Life-threatening |
Which leads should be evaluated?
The catalog marks as primary diagnostic leads II, V1, V6. Rapid wide-complex tachycardia with continuously varying QRS morphology.
In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.
Frequently asked clinical questions
How is Polymorphic Ventricular Tachycardia identified on the 12-lead ECG?
Ventricular tachycardia with beat-to-beat variation in QRS morphology and axis. Commonly triggered by acute myocardial ischemia (coronary occlusion), severe electrolyte abnormalities, or catecholaminergic surge. Rapidly deteriorates into ventricular fibrillation and hemodynamic collapse. The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.
Which leads are critical for diagnostic confirmation?
The primary diagnostic leads are II, V1, V6. Rapid wide-complex tachycardia with continuously varying QRS morphology. In the simulator, these channels highlight the correlation with anatomical vectors.
What is the underlying electrophysiological or anatomical mechanism?
It originates from altered conduction in the cardiac conduction system, mediated by reentrada.
How can this condition be explored in the 3D simulator?
MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.
How do you open this configuration?
The simulator encodes its full state in the query string, providing a stable and reproducible URL:
https://mioatlas.com/simulador/en/?mode=conduction&arr=VT_POLYMORPHIC
Related entities
Other entities in the same category within the catalog:
Sources and bibliographic references
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Anderson RH, Yanni J, Boyett MR, Chandler NJ, Dobrzynski H (2009). "The anatomy of the cardiac conduction system". Clin Anat.
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Rautaharju PM, Surawicz B, Gettes LS, et al. (2009). "AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram: Part IV: The ST segment, T and U waves, and the QT interval". Circulation.
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Kligfield P, Gettes LS, Bailey JJ, et al. (2007). "Recommendations for the standardization and interpretation of the electrocardiogram: Part I: The electrocardiogram and its technology". Circulation.