PVC
Technical documentation
Premature Ventricular Complexes
Premature ectopic impulse originating in ventricular myocardium. QRS is wide (≥ 120 ms) and bizarrely deformed, with T wave and ST segment directed opposite to the main QRS deflection. No preceding P wave; followed by a full compensatory pause.
Also known as: Premature Ventricular Complexes · extrasistoles ventriculares · extrasistole ventricular · complejos ventriculares prematuros · latidos ventriculares prematuros · premature ventricular contraction · PVC · EV · VPB
What parameters does the model use?
The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.
What parameters does the model use?
| Parameter | Model value |
|---|---|
| Mechanism | Automatismo |
| Impulse origin | Sinoatrial node (SA node) |
| Modeled rate | 60–100 bpm (default 75) |
| Regularity | Irregular |
| P wave | Sinus (upright in I, II, aVF) |
| PR interval | 120–200 ms (default 160) |
| QRS duration | 90 ms |
| QRS morphology | Normal (narrow) |
| AV conduction | 1:1 AV conduction |
| Severity | Benign |
Which leads should be evaluated?
The catalog marks as primary diagnostic leads V1, V6, II. Premature wide bizarre QRS complex without preceding P wave and discordant T wave.
In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.
Frequently asked clinical questions
How is Premature Ventricular Complexes identified on the 12-lead ECG?
Premature ectopic impulse originating in ventricular myocardium. QRS is wide (≥ 120 ms) and bizarrely deformed, with T wave and ST segment directed opposite to the main QRS deflection. No preceding P wave; followed by a full compensatory pause. The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.
Which leads are critical for diagnostic confirmation?
The primary diagnostic leads are V1, V6, II. Premature wide bizarre QRS complex without preceding P wave and discordant T wave. In the simulator, these channels highlight the correlation with anatomical vectors.
What is the underlying electrophysiological or anatomical mechanism?
It originates from altered conduction in the SA_NODE, mediated by automatismo.
How can this condition be explored in the 3D simulator?
MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.
How do you open this configuration?
The simulator encodes its full state in the query string, providing a stable and reproducible URL:
https://mioatlas.com/simulador/en/?mode=conduction&arr=PVC
Related entities
Other entities in the same category within the catalog:
Sources and bibliographic references
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Anderson RH, Yanni J, Boyett MR, Chandler NJ, Dobrzynski H (2009). "The anatomy of the cardiac conduction system". Clin Anat.
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Rautaharju PM, Surawicz B, Gettes LS, et al. (2009). "AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram: Part IV: The ST segment, T and U waves, and the QT interval". Circulation.
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Kligfield P, Gettes LS, Bailey JJ, et al. (2007). "Recommendations for the standardization and interpretation of the electrocardiogram: Part I: The electrocardiogram and its technology". Circulation.