TORSADES
Technical documentation
Torsade de Pointes
Specific form of polymorphic ventricular tachycardia occurring in the setting of prolonged baseline QT interval (congenital or drug-induced). QRS complexes appear to twist their polarity and amplitude around the isoelectric baseline in an undulating fashion. Often triggered by an R-on-T premature ventricular beat after a pause ('short-long-short' sequence).
Also known as: Torsade de Pointes · torsade de pointes · torsades · torsades de pointes · torsade de puntas · TdP · taquicardia helicoidal · QT largo con torsade
What parameters does the model use?
The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.
What parameters does the model use?
| Parameter | Model value |
|---|---|
| Mechanism | Reentrada |
| Modeled rate | 200–280 bpm (default 240) |
| Regularity | Irregular |
| P wave | Dissociated from QRS |
| QRS duration | 180 ms |
| QRS morphology | Twisting of polarity around baseline |
| AV conduction | Atrioventricular dissociation |
| Severity | Life-threatening |
Which leads should be evaluated?
The catalog marks as primary diagnostic leads II, V1, V6. QRS peaks twisting around the isoelectric baseline in the setting of prolonged QT.
In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.
Frequently asked clinical questions
How is Torsade de Pointes identified on the 12-lead ECG?
Specific form of polymorphic ventricular tachycardia occurring in the setting of prolonged baseline QT interval (congenital or drug-induced). QRS complexes appear to twist their polarity and amplitude around the isoelectric baseline in an undulating fashion. Often triggered by an R-on-T premature ventricular beat after a pause ('short-long-short' sequence). The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.
Which leads are critical for diagnostic confirmation?
The primary diagnostic leads are II, V1, V6. QRS peaks twisting around the isoelectric baseline in the setting of prolonged QT. In the simulator, these channels highlight the correlation with anatomical vectors.
What is the underlying electrophysiological or anatomical mechanism?
It originates from altered conduction in the cardiac conduction system, mediated by reentrada.
How can this condition be explored in the 3D simulator?
MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.
How do you open this configuration?
The simulator encodes its full state in the query string, providing a stable and reproducible URL:
https://mioatlas.com/simulador/en/?mode=conduction&arr=TORSADES
Related entities
Other entities in the same category within the catalog:
Sources and bibliographic references
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Fridericia LS (1920). "Die Systolendauer im Elektrokardiogramm bei normalen Menschen und bei Herzkranken". Acta Med Scand.
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Anderson RH, Yanni J, Boyett MR, Chandler NJ, Dobrzynski H (2009). "The anatomy of the cardiac conduction system". Clin Anat.
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Rautaharju PM, Surawicz B, Gettes LS, et al. (2009). "AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram: Part IV: The ST segment, T and U waves, and the QT interval". Circulation.
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Kligfield P, Gettes LS, Bailey JJ, et al. (2007). "Recommendations for the standardization and interpretation of the electrocardiogram: Part I: The electrocardiogram and its technology". Circulation.