AIVR
Technical documentation
Accelerated Idioventricular Rhythm
Ventricular rhythm at 50-100 bpm (faster than ventricular escape, slower than ventricular tachycardia). Most commonly seen as a benign sign of successful myocardial reperfusion after primary PCI or thrombolysis in acute STEMI. Usually well tolerated, self-limiting, and requires no suppressive antiarrhythmic therapy.
Also known as: Accelerated Idioventricular Rhythm · ritmo idioventricular acelerado · RIVA · accelerated idioventricular rhythm · AIVR · ritmo de reperfusion · slow VT
What parameters does the model use?
The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.
What parameters does the model use?
| Parameter | Model value |
|---|---|
| Mechanism | Automatismo |
| Modeled rate | 60–110 bpm (default 85) |
| Regularity | Regular |
| P wave | Dissociated from QRS |
| QRS duration | 150 ms |
| QRS morphology | Wide, ventricular origin |
| AV conduction | Atrioventricular dissociation |
| Severity | Benign |
Which leads should be evaluated?
The catalog marks as primary diagnostic leads V1, V6, II. Regular wide-complex rhythm at 60-110 bpm with frequent AV dissociation.
In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.
Frequently asked clinical questions
How is Accelerated Idioventricular Rhythm identified on the 12-lead ECG?
Ventricular rhythm at 50-100 bpm (faster than ventricular escape, slower than ventricular tachycardia). Most commonly seen as a benign sign of successful myocardial reperfusion after primary PCI or thrombolysis in acute STEMI. Usually well tolerated, self-limiting, and requires no suppressive antiarrhythmic therapy. The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.
Which leads are critical for diagnostic confirmation?
The primary diagnostic leads are V1, V6, II. Regular wide-complex rhythm at 60-110 bpm with frequent AV dissociation. In the simulator, these channels highlight the correlation with anatomical vectors.
What is the underlying electrophysiological or anatomical mechanism?
It originates from altered conduction in the cardiac conduction system, mediated by automatismo.
How can this condition be explored in the 3D simulator?
MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.
How do you open this configuration?
The simulator encodes its full state in the query string, providing a stable and reproducible URL:
https://mioatlas.com/simulador/en/?mode=conduction&arr=AIVR
Related entities
Other entities in the same category within the catalog:
Sources and bibliographic references
-
Anderson RH, Yanni J, Boyett MR, Chandler NJ, Dobrzynski H (2009). "The anatomy of the cardiac conduction system". Clin Anat.
-
Rautaharju PM, Surawicz B, Gettes LS, et al. (2009). "AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram: Part IV: The ST segment, T and U waves, and the QT interval". Circulation.
-
Kligfield P, Gettes LS, Bailey JJ, et al. (2007). "Recommendations for the standardization and interpretation of the electrocardiogram: Part I: The electrocardiogram and its technology". Circulation.