AVB_2_MOBITZ_I
Technical documentation
Second-Degree AV Block Mobitz I (Wenckebach)
Progressive prolongation of the PR interval in successive beats until a P wave fails to conduct (blocked P wave). The pause that follows is shorter than two normal cycles, and the cycle then restarts. Almost always located within the AV node; usually benign and reversible (vagal hypertonia, inferior STEMI, drugs).
Also known as: Second-Degree AV Block Mobitz I (Wenckebach) · Mobitz I · Wenckebach · bloqueo AV de 2.º grado Mobitz I · bloqueo AV Mobitz I · BAV 2 Mobitz I · BAV Mobitz I · bloqueo de Wenckebach · bloqueo AV de segundo grado tipo I · second-degree AV block type I · Mobitz type I
What parameters does the model use?
The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.
What parameters does the model use?
| Parameter | Model value |
|---|---|
| Mechanism | Bloqueo |
| Impulse origin | Sinoatrial node (SA node) |
| Modeled rate | 50–90 bpm (default 68) |
| Regularity | Regularly irregular |
| P wave | Sinus (upright in I, II, aVF) |
| PR interval | 170–320 ms (default 220) |
| QRS duration | 90 ms |
| QRS morphology | Normal (narrow) |
| AV conduction | Wenckebach (progressive PR lengthening) |
| Severity | Benign |
Which leads should be evaluated?
The catalog marks as primary diagnostic leads II. Progressive PR prolongation until a P wave fails to conduct (Wenckebach) in lead II.
In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.
Frequently asked clinical questions
How is Second-Degree AV Block Mobitz I (Wenckebach) identified on the 12-lead ECG?
Progressive prolongation of the PR interval in successive beats until a P wave fails to conduct (blocked P wave). The pause that follows is shorter than two normal cycles, and the cycle then restarts. Almost always located within the AV node; usually benign and reversible (vagal hypertonia, inferior STEMI, drugs). The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.
Which leads are critical for diagnostic confirmation?
The primary diagnostic leads are II. Progressive PR prolongation until a P wave fails to conduct (Wenckebach) in lead II. In the simulator, these channels highlight the correlation with anatomical vectors.
What is the underlying electrophysiological or anatomical mechanism?
It originates from altered conduction in the SA_NODE, mediated by bloqueo.
How can this condition be explored in the 3D simulator?
MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.
How do you open this configuration?
The simulator encodes its full state in the query string, providing a stable and reproducible URL:
https://mioatlas.com/simulador/en/?mode=conduction&arr=AVB_2_MOBITZ_I
Related entities
Other entities in the same category within the catalog:
Related clinical comparisons
Sources and bibliographic references
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Anderson RH, Yanni J, Boyett MR, Chandler NJ, Dobrzynski H (2009). "The anatomy of the cardiac conduction system". Clin Anat.
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Kligfield P, Gettes LS, Bailey JJ, et al. (2007). "Recommendations for the standardization and interpretation of the electrocardiogram: Part I: The electrocardiogram and its technology". Circulation.
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Surawicz B, Childers R, Deal BJ, et al. (2009). "AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram: Part III: Intraventricular conduction disturbances". Circulation.