AVB_2_1
Technical documentation
AV Block 2:1
Every other P wave is blocked in the AV conduction system: one P conducts, the next is blocked. Because two consecutive conducted PR intervals are never seen, it cannot be categorized as Mobitz I or II by PR behavior alone. High-grade AV block requiring careful assessment of QRS width (narrow = likely nodal; wide = likely infranodal).
Also known as: 2:1 AV Block · bloqueo AV 2:1 · BAV 2:1 · bloqueo auriculoventricular 2:1 · 2:1 AV block · 2 to 1 AV block
What parameters does the model use?
The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.
What parameters does the model use?
| Parameter | Model value |
|---|---|
| Mechanism | Bloqueo |
| Impulse origin | Sinoatrial node (SA node) |
| Modeled rate | 30–50 bpm (default 40) |
| Regularity | Regular |
| P wave | Sinus (upright in I, II, aVF) |
| PR interval | 160–240 ms (default 200) |
| QRS duration | 100 ms |
| QRS morphology | Normal (narrow) |
| AV conduction | 2:1 AV block |
| Severity | Clinically significant |
Which leads should be evaluated?
The catalog marks as primary diagnostic leads II. Every other P wave is blocked with regular 2:1 ratio in lead II.
In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.
Frequently asked clinical questions
How is 2:1 AV Block identified on the 12-lead ECG?
Every other P wave is blocked in the AV conduction system: one P conducts, the next is blocked. Because two consecutive conducted PR intervals are never seen, it cannot be categorized as Mobitz I or II by PR behavior alone. High-grade AV block requiring careful assessment of QRS width (narrow = likely nodal; wide = likely infranodal). The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.
Which leads are critical for diagnostic confirmation?
The primary diagnostic leads are II. Every other P wave is blocked with regular 2:1 ratio in lead II. In the simulator, these channels highlight the correlation with anatomical vectors.
What is the underlying electrophysiological or anatomical mechanism?
It originates from altered conduction in the SA_NODE, mediated by bloqueo.
How can this condition be explored in the 3D simulator?
MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.
How do you open this configuration?
The simulator encodes its full state in the query string, providing a stable and reproducible URL:
https://mioatlas.com/simulador/en/?mode=conduction&arr=AVB_2_1
Related entities
Other entities in the same category within the catalog:
Sources and bibliographic references
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Anderson RH, Yanni J, Boyett MR, Chandler NJ, Dobrzynski H (2009). "The anatomy of the cardiac conduction system". Clin Anat.
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Kligfield P, Gettes LS, Bailey JJ, et al. (2007). "Recommendations for the standardization and interpretation of the electrocardiogram: Part I: The electrocardiogram and its technology". Circulation.
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Surawicz B, Childers R, Deal BJ, et al. (2009). "AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram: Part III: Intraventricular conduction disturbances". Circulation.