Atrioventricular Blocks AVB_2_MOBITZ_II Technical documentation

Second-Degree AV Block Mobitz II

Sudden failure of AV conduction with an intermittent dropped P wave WITHOUT prior PR interval prolongation. The block is located in the His bundle or bundle branches (infranodal). High risk of progression to complete infrahisian block and Stokes-Adams syncope; absolute indication for permanent pacemaker implantation.

Generated from the computational engine catalog · Educational reference without specialist cardiology review.

Also known as: Second-Degree AV Block Mobitz II · Mobitz II · bloqueo AV de 2.º grado Mobitz II · bloqueo AV Mobitz II · BAV 2 Mobitz II · BAV Mobitz II · bloqueo auriculoventricular tipo Mobitz II · bloqueo AV de segundo grado tipo II · second-degree AV block type II · Mobitz type II · second degree AV block Mobitz II

What parameters does the model use?

The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.

What parameters does the model use?

Parameter Model value
Mechanism Bloqueo
Impulse origin Sinoatrial node (SA node)
Modeled rate 40–75 bpm (default 55)
Regularity Regularly irregular
P wave Sinus (upright in I, II, aVF)
PR interval 150–200 ms (default 180)
QRS duration 130 ms
QRS morphology Right bundle branch block morphology
AV conduction Variable
Severity Life-threatening

Which leads should be evaluated?

The catalog marks as primary diagnostic leads II, V1. Intermittent dropped P waves with constant preceding PR intervals; wide QRS in V1.

In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.

Frequently asked clinical questions

How is Second-Degree AV Block Mobitz II identified on the 12-lead ECG?

Sudden failure of AV conduction with an intermittent dropped P wave WITHOUT prior PR interval prolongation. The block is located in the His bundle or bundle branches (infranodal). High risk of progression to complete infrahisian block and Stokes-Adams syncope; absolute indication for permanent pacemaker implantation. The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.

Which leads are critical for diagnostic confirmation?

The primary diagnostic leads are II, V1. Intermittent dropped P waves with constant preceding PR intervals; wide QRS in V1. In the simulator, these channels highlight the correlation with anatomical vectors.

What is the underlying electrophysiological or anatomical mechanism?

It originates from altered conduction in the SA_NODE, mediated by bloqueo.

How can this condition be explored in the 3D simulator?

MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.

How do you open this configuration?

The simulator encodes its full state in the query string, providing a stable and reproducible URL:

https://mioatlas.com/simulador/en/?mode=conduction&arr=AVB_2_MOBITZ_II

Related entities

Other entities in the same category within the catalog:

Related clinical comparisons

Sources and bibliographic references

  1. Anderson RH, Yanni J, Boyett MR, Chandler NJ, Dobrzynski H (2009). "The anatomy of the cardiac conduction system". Clin Anat.
  2. Kligfield P, Gettes LS, Bailey JJ, et al. (2007). "Recommendations for the standardization and interpretation of the electrocardiogram: Part I: The electrocardiogram and its technology". Circulation.
  3. Surawicz B, Childers R, Deal BJ, et al. (2009). "AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram: Part III: Intraventricular conduction disturbances". Circulation.