AVB_3_INFRAHISIAN
Technical documentation
Complete Infrahisian AV Block
Total interruption of AV conduction located below the His bundle (in both bundle branches or all three fascicles). Accompanied by a low, wide, unstable ventricular escape pacemaker at 20-40 bpm. Severe hemodynamic compromise with syncope, heart failure, and immediate risk of asystole.
Also known as: Complete Infrahisian AV Block · bloqueo AV completo infrahisiano · bloqueo infrahisiano · BAV completo infrahisiano · bloqueo auriculoventricular infrahisiano · infra-Hisian block · infrahisian AV block
What parameters does the model use?
The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.
What parameters does the model use?
| Parameter | Model value |
|---|---|
| Mechanism | Bloqueo |
| Modeled rate | 20–40 bpm (default 32) |
| Regularity | Regular |
| P wave | Dissociated from QRS |
| QRS duration | 165 ms |
| QRS morphology | Wide, ventricular origin |
| AV conduction | Atrioventricular dissociation |
| Severity | Life-threatening |
Which leads should be evaluated?
The catalog marks as primary diagnostic leads II, V1. Infrahisian block: dissociated P waves with very slow, wide sub-Hisian escape QRS.
In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.
Frequently asked clinical questions
How is Complete Infrahisian AV Block identified on the 12-lead ECG?
Total interruption of AV conduction located below the His bundle (in both bundle branches or all three fascicles). Accompanied by a low, wide, unstable ventricular escape pacemaker at 20-40 bpm. Severe hemodynamic compromise with syncope, heart failure, and immediate risk of asystole. The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.
Which leads are critical for diagnostic confirmation?
The primary diagnostic leads are II, V1. Infrahisian block: dissociated P waves with very slow, wide sub-Hisian escape QRS. In the simulator, these channels highlight the correlation with anatomical vectors.
What is the underlying electrophysiological or anatomical mechanism?
It originates from altered conduction in the cardiac conduction system, mediated by bloqueo.
How can this condition be explored in the 3D simulator?
MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.
How do you open this configuration?
The simulator encodes its full state in the query string, providing a stable and reproducible URL:
https://mioatlas.com/simulador/en/?mode=conduction&arr=AVB_3_INFRAHISIAN
Related entities
Other entities in the same category within the catalog:
Sources and bibliographic references
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Anderson RH, Yanni J, Boyett MR, Chandler NJ, Dobrzynski H (2009). "The anatomy of the cardiac conduction system". Clin Anat.
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Kligfield P, Gettes LS, Bailey JJ, et al. (2007). "Recommendations for the standardization and interpretation of the electrocardiogram: Part I: The electrocardiogram and its technology". Circulation.
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Surawicz B, Childers R, Deal BJ, et al. (2009). "AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram: Part III: Intraventricular conduction disturbances". Circulation.