Left Main Coronary Artery (LMCA) LMCA Technical documentation

Left Main Coronary Artery (LMCA)

Involvement of the left main trunk before bifurcation. Compromises the entirety of the left ventricle (LAD + LCx). Cardiogenic shock and extreme risk. ST elevation in aVR >= V1 with deep diffuse multilead depression (I, II, aVL, V4-V6).

Generated from the computational engine catalog · Educational reference without specialist cardiology review.

Also known as: Left Main Coronary Artery (LMCA) · tronco coronario izquierdo · TCI · oclusion de tronco · infarto de tronco · oclusión del tronco coronario izquierdo · left main · LMCA · left main coronary artery · left main stem · left main STEMI

What parameters does the model use?

The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.

What parameters does the model use?

Parameter Model value
Involved artery LMCA
ST elevation in aVR, V1, V2
Reciprocal depression in I, II, aVL, V4, V5, V6
AHA 17-segment model 1, 2, 3, 4, 5, 6, 7, 8, 9, 10, 11, 12, 13, 14, 15, 16, 17
Occlusion severity Total

Which leads should be evaluated?

The catalog marks as primary diagnostic leads aVR, V1, V2. ST elevation in aVR, V1, V2; reciprocal ST depression in I, II, aVL, V4, V5, V6.

In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.

Frequently asked clinical questions

How is Left Main Coronary Artery (LMCA) identified on the 12-lead ECG?

Involvement of the left main trunk before bifurcation. Compromises the entirety of the left ventricle (LAD + LCx). Cardiogenic shock and extreme risk. ST elevation in aVR >= V1 with deep diffuse multilead depression (I, II, aVL, V4-V6). The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.

Which leads are critical for diagnostic confirmation?

The primary diagnostic leads are aVR, V1, V2. ST elevation in aVR, V1, V2; reciprocal ST depression in I, II, aVL, V4, V5, V6. In the simulator, these channels highlight the correlation with anatomical vectors.

What is the underlying electrophysiological or anatomical mechanism?

It represents transmural ischemia secondary to occlusion in the territory of the LMCA.

How can this condition be explored in the 3D simulator?

MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.

How do you open this configuration?

The simulator encodes its full state in the query string, providing a stable and reproducible URL:

https://mioatlas.com/simulador/en/?syn=SCACEST&ter=LMCA

Sources and bibliographic references

  1. Rao SV, O'Donoghue ML, Ruel M, et al. (2025). "2025 ACC/AHA/ACEP/NAEMSP/SCAI Guideline for the Management of Patients With Acute Coronary Syndromes". Circulation.
  2. Wagner GS, Macfarlane P, Wellens H, et al. (2009). "AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram: Part VI: Acute ischemia/infarction". Circulation.
  3. Cerqueira MD, Weissman NJ, Dilsizian V, et al. (2002). "Standardized myocardial segmentation and nomenclature for tomographic imaging of the heart". Circulation.
  4. Thygesen K, Alpert JS, Jaffe AS, et al. (2019). "Fourth Universal Definition of Myocardial Infarction (2018)". Eur Heart J.