Intraventricular Blocks LBBB Technical documentation

Complete Left Bundle Branch Block

Complete block in the main trunk of the left bundle branch. Ventricular activation is drastically altered: the septum activates from right to left, and the left ventricle activates late and slowly. Wide QRS (≥ 120 ms), broad notched or slurred R waves in I, aVL, V5-V6, and deep QS or rS in V1-V2. Normal septal q waves in lateral leads disappear.

Generated from the computational engine catalog · Educational reference without specialist cardiology review.

Also known as: Complete Left Bundle Branch Block · bloqueo de rama izquierda · BRI · bloqueo completo de rama izquierda · bloqueo de rama izquierda del haz de His · left bundle branch block · LBBB · complete LBBB

What parameters does the model use?

The following values are applied by the simulation engine to this entity. They govern activation kinematics in the 3D model and the real-time synthesis of the 12-lead electrocardiogram.

What parameters does the model use?

Parameter Model value
Mechanism Bloqueo
Impulse origin Sinoatrial node (SA node)
Modeled rate 60–100 bpm (default 75)
Regularity Regular
P wave Sinus (upright in I, II, aVF)
PR interval 120–200 ms (default 160)
QRS duration 150 ms
QRS morphology Left bundle branch block morphology
AV conduction 1:1 AV conduction
Severity Clinically significant

Which leads should be evaluated?

The catalog marks as primary diagnostic leads V1, V2, V3, I, aVL, V5, V6. Deep QS or rS in V1-V3; broad notched R wave without septal q in leads I, aVL, V5-V6.

In the simulator, these leads are highlighted on the 12-channel tracing and corresponding vectors are rendered on the 3D model, displaying electrical changes and anatomical origin simultaneously.

Frequently asked clinical questions

How is Complete Left Bundle Branch Block identified on the 12-lead ECG?

Complete block in the main trunk of the left bundle branch. Ventricular activation is drastically altered: the septum activates from right to left, and the left ventricle activates late and slowly. Wide QRS (≥ 120 ms), broad notched or slurred R waves in I, aVL, V5-V6, and deep QS or rS in V1-V2. Normal septal q waves in lateral leads disappear. The characteristic diagnostic waveforms are generated in real time across the simulated 12-lead tracing.

Which leads are critical for diagnostic confirmation?

The primary diagnostic leads are V1, V2, V3, I, aVL, V5, V6. Deep QS or rS in V1-V3; broad notched R wave without septal q in leads I, aVL, V5-V6. In the simulator, these channels highlight the correlation with anatomical vectors.

What is the underlying electrophysiological or anatomical mechanism?

It originates from altered conduction in the SA_NODE, mediated by bloqueo.

How can this condition be explored in the 3D simulator?

MioAtlas offers direct deep-link reproducibility with preconfigured parameters, allowing full 3D cardiac rotation and lead vector inspection.

How do you open this configuration?

The simulator encodes its full state in the query string, providing a stable and reproducible URL:

https://mioatlas.com/simulador/en/?mode=conduction&arr=LBBB

Related entities

Other entities in the same category within the catalog:

Related clinical comparisons

Sources and bibliographic references

  1. Surawicz B, Childers R, Deal BJ, et al. (2009). "AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram: Part III: Intraventricular conduction disturbances". Circulation.
  2. Anderson RH, Yanni J, Boyett MR, Chandler NJ, Dobrzynski H (2009). "The anatomy of the cardiac conduction system". Clin Anat.
  3. Kligfield P, Gettes LS, Bailey JJ, et al. (2007). "Recommendations for the standardization and interpretation of the electrocardiogram: Part I: The electrocardiogram and its technology". Circulation.