Atlante coronarico e ischemia 3D

Simulatore STEMI 3D: territori coronarici ed equivalenti OMI

Visualizza le ripercussioni bioelettriche e tissutali dell'occlusione coronarica acuta su un cuore 3D interattivo con derivazioni standard e accessorie.

Coronary tree modeling & myocardial perfusion

The MioAtlas coronary engine reconstructs the three-dimensional architecture of the 11 major arterial branches over the ventricular epicardium: left main (LMCA), left anterior descending (LAD) with D1 and D2 diagonals, circumflex (LCx) with OM1 and OM2 marginals, and right coronary artery (RCA) with acute marginal (AM), posterior descending (PDA), and posterolateral (PLB) branches.

Each stenosis or occlusion point (parameter ter=...) defines an ischemic myocardium-at-risk volume based on vessel location and coronary dominance. Transmural injury is mapped onto the standardized AHA/ACC 17-segment model (Cerqueira et al. 2002). Cellular injury currents during depolarization and repolarization yield ST-vector shifts recorded on the live 120 Hz ECG monitor.

OMI paradigm: beyond strict millimeter criteria

The simulator integrates 8 ECG phenotypes reflecting the modern Occlusion Myocardial Infarction (OMI) paradigm. This framework recognizes that many acute transmural or critical subtotal occlusions do not meet traditional millimeter ST elevation thresholds in contiguous leads.

Modeled patterns include De Winter hyperacute T-waves in critical proximal LAD stenosis, Wellens biphasic/inverted T-wave reperfusion patterns (Types A and B), Aslanger inferior infarction with multivessel disease, giant Shark-Fin triangular waves, and modified Sgarbossa-Smith criteria in LBBB and ventricular pacing.

Biophysical variables: evolutionary phases & coronary dominance

ECG manifestations change dynamically with ischemic duration and coronary anatomy. The engine allows interactive selection of infarction phases (ph=HIPERAGUDA, AGUDA, ESTABLECIDA, SUBAGUDA, or CRONICA), demonstrating the progression from hyperacute T-waves to pathological Q-wave necrosis (>30 ms) and T-wave inversion.

Dominance settings (dom=RIGHT, LEFT, CODOMINANT) redistribute vascular supply to the inferior and posterior LV, altering whether maximum ST elevation appears in lead III (right dominant) or lead II with inferolateral involvement (left dominant).

Esplorare l'albero coronarico in 3D

Avvia il simulatore in modalità coronarica con infarto della discendente anteriore e modifica territorio, dominanza o fase evolutiva.

Apri simulatore coronarico 3D

18 Siti di occlusione coronarica (STEMI / Infarto transmurale)

Localizzazioni anatomiche sul tronco comune, discendente anteriore, circonflessa, coronaria destra e rami secondari.

Tronco Comune Sinistro (TCS / LMCA)

1 location

Discendente Anteriore (IVA / LAD)

5 locations

Circonflessa (Cx / LCx)

2 locations

Coronaria Destra (CD / RCA)

4 locations

Rami Coronarici (Diagonali, Marginali, PDA, PLB)

6 locations
Paradigma OMI

8 Fenotipi elettrocardiografici ed equivalenti OMI

Pattern ischemici ad alto rischio indicativi di occlusione coronarica acuta senza sopraslivellamento ST classico.

Hemodynamic and electrophysiological foundation

The 11-artery coronary model and myocardium-at-risk mapping build upon standardized American Heart Association anatomical classifications (Cerqueira et al. 2002; Austen et al. 1975). Culprit artery correlation and reciprocal leads follow European Society of Cardiology and American College of Cardiology consensus guidelines (Thygesen et al. 2018; Ibanez et al. 2018).